The reduction in phosphocreatine depletion and in the increase in ADP during exercise, and the enhanced rate of phosphocreatine resynthesis in recovery (which is independent of muscle mass) indicate that a substantial correction of the impaired oxidative capacity of skeletal muscle in chronic heart failure can be achieved by exercise training.
Pedro Ángel Colao MarínMaría del Carmen Pastor del PinoSuardíaz Muro, Juan
Pedro Ángel Colao MarínMaría Carmen Pastor del Pino
Colao Marín, Pedro ÁngelPastor del Pino, María del Carmen
Gabriel Muñoz MartínezJuan Zornoza Pérez