Syndecan-1 regulates extracellular matrix expression in keloid fibroblasts via TGF-β1/Smad and MAPK signaling pathways

Jing Cui; Shan Jin; Chenglong Jin; Zhehu Jin

doi:10.1016/j.lfs.2020.117326

ScienceGate Book Chapters

JOURNAL ARTICLE

Syndecan-1 regulates extracellular matrix expression in keloid fibroblasts via TGF-β1/Smad and MAPK signaling pathways

Jing Cui Shan Jin Chenglong Jin Zhehu Jin

Year: 2020 Journal: Life Sciences Vol: 254 Pages: 117326-117326 Publisher: Elsevier BV

DOI: 10.1016/j.lfs.2020.117326

Get Full-Text PDF Get Analytical Report

Keywords:

Keloid Syndecan 1 SMAD Extracellular matrix MAPK/ERK pathway Western blot Fibroblast Signal transduction Extracellular Chemistry Cell growth Molecular biology Cell biology Cancer research Transfection Transforming growth factor Biology Cell Medicine Pathology Biochemistry In vitro

Metrics

Cited By

4.17

FWCI (Field Weighted Citation Impact)

Refs

0.92

Citation Normalized Percentile

Is in top 1%

Is in top 10%

Citation History

Topics

Dermatologic Treatments and Research

Health Sciences → Medicine → Dermatology

Wound Healing and Treatments

Health Sciences → Medicine → Rehabilitation

Connective Tissue Growth Factor Research

Life Sciences → Biochemistry, Genetics and Molecular Biology → Molecular Biology

Syndecan-1 regulates extracellular matrix expression in keloid fibroblasts via TGF-β1/Smad and MAPK signaling pathways

Metrics

Citation History

Topics

Related Documents

Carvacrol Inhibits the Proliferation and Extracellular Matrix Deposition of Keloid Fibroblasts Through Nrf2/GPX4 and TGF-β1/Smad Signaling Pathways

Oleanolic acid regulates the proliferation and extracellular matrix of keloid fibroblasts by mediating the TGF‐β1/SMAD signaling pathway

TGF-β1-induced HSP47 regulates extracellular matrix accumulation via Smad2/3 signaling pathways in nasal fibroblasts

Knockdown of elF3a inhibits TGF‑β1‑induced extracellular matrix protein expression in keloid fibroblasts

DKK3 regulates cell proliferation, apoptosis and collagen synthesis in keloid fibroblasts via TGF-β1/Smad signaling pathway