Inhaled Nitric Oxide

George F. Rich; Gerald D. Murphy; C. M. Roos; Roger A. Johns

doi:10.1097/00000542-199306000-00004

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JOURNAL ARTICLE

Inhaled Nitric Oxide

George F. Rich Gerald D. Murphy C. M. Roos Roger A. Johns

Year: 1993 Journal: Anesthesiology Vol: 78 (6)Pages: 1028-1035 Publisher: Lippincott Williams & Wilkins

DOI: 10.1097/00000542-199306000-00004

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Abstract

This study demonstrates that 20 ppm inhaled NO is a selective pulmonary vasodilator in cardiac surgical patients before and after CPB and in patients in whom the circulation is supported with a VAD. Furthermore, NO-induced pulmonary vasodilation is proportional to PVRb and does not appear to be altered by CPB, the presence of a VAD, or infusion of nitrates.

Keywords:

Medicine Vascular resistance Pulmonary artery Anesthesia Hemodynamics Cardiopulmonary bypass Cardiac output Nitric oxide Pulmonary hypertension Cardiology Inhalation Internal medicine Central venous pressure Vasodilation Blood pressure Heart rate

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Pulmonary Hypertension Research and Treatments

Health Sciences → Medicine → Pulmonary and Respiratory Medicine

Cardiac, Anesthesia and Surgical Outcomes

Health Sciences → Medicine → Cardiology and Cardiovascular Medicine

Respiratory Support and Mechanisms

Health Sciences → Medicine → Pulmonary and Respiratory Medicine

Inhaled Nitric Oxide

Abstract

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